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Wright, S. C., Bonavida, B. (1983) YAC-1 variant clones selected for resistance to natural killer cytotoxic factors are also resistant to natural killer cell-mediated cytotoxicity. Proceedings of the National Academy of Sciences, 80 (6) 1688-1692 doi:10.1073/pnas.80.6.1688

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Reference TypeJournal (article/letter/editorial)
TitleYAC-1 variant clones selected for resistance to natural killer cytotoxic factors are also resistant to natural killer cell-mediated cytotoxicity.
JournalProceedings of the National Academy of Sciences
AuthorsWright, S. C.Author
Bonavida, B.Author
Year1983 (March 1)Volume80
Issue6
PublisherProceedings of the National Academy of Sciences
DOIdoi:10.1073/pnas.80.6.1688Search in ResearchGate
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Mindat Ref. ID1433101Long-form Identifiermindat:1:5:1433101:7
GUID0
Full ReferenceWright, S. C., Bonavida, B. (1983) YAC-1 variant clones selected for resistance to natural killer cytotoxic factors are also resistant to natural killer cell-mediated cytotoxicity. Proceedings of the National Academy of Sciences, 80 (6) 1688-1692 doi:10.1073/pnas.80.6.1688
Plain TextWright, S. C., Bonavida, B. (1983) YAC-1 variant clones selected for resistance to natural killer cytotoxic factors are also resistant to natural killer cell-mediated cytotoxicity. Proceedings of the National Academy of Sciences, 80 (6) 1688-1692 doi:10.1073/pnas.80.6.1688
In(1983, March) Proceedings of the National Academy of Sciences Vol. 80 (6) Proceedings of the National Academy of Sciences

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Lebow, L. T., Bonavida, B. (1990) Purification and characterization of cytolytic and noncytolytic human natural killer cell subsets. Proceedings of the National Academy of Sciences, 87 (16) 6063-6067 doi:10.1073/pnas.87.16.6063
BONAVIDA, BENJAMIN (1988) Mechanism of Natural Killer Cell-Mediated Cytotoxicity. Annals of the New York Academy of Sciences, 532 (1) 336-340 doi:10.1111/j.1749-6632.1988.tb36350.x
Zucker-Franklin, D., Grusky, G., Yang, J. S. (1983) Arylsulfatase in natural killer cells: its possible role in cytotoxicity. Proceedings of the National Academy of Sciences, 80 (22) 6977-6981 doi:10.1073/pnas.80.22.6977
Burns, G. F., Triglia, T., Bartlett, P. F., Mackay, I. R. (1983) Human natural killer cells, activated lymphocyte killer cells, and monocytes possess similar cytotoxic mechanisms. Proceedings of the National Academy of Sciences, 80 (24) 7606-7610 doi:10.1073/pnas.80.24.7606
Roder, J. C., Beaumont, T. J., Kerbel, R. S., Haliotis, T., Kozbor, D. (1981) Selective natural killer resistance in a clone of YAC lymphoma cells. Proceedings of the National Academy of Sciences, 78 (10) 6396-6400 doi:10.1073/pnas.78.10.6396
Kim, S., Iizuka, K., Aguila, H. L., Weissman, I. L., Yokoyama, W. M. (2000) In vivo natural killer cell activities revealed by natural killer cell-deficient mice. Proceedings of the National Academy of Sciences, 97 (6) 2731-2736 doi:10.1073/pnas.050588297
Lowin, B., Beermann, F., Schmidt, A., Tschopp, J. (1994) A null mutation in the perforin gene impairs cytolytic T lymphocyte- and natural killer cell-mediated cytotoxicity. Proceedings of the National Academy of Sciences, 91 (24) 11571-11575 doi:10.1073/pnas.91.24.11571
Biron, C. A. (2012) Yet another role for natural killer cells: Cytotoxicity in immune regulation and viral persistence. Proceedings of the National Academy of Sciences, 109 (6) 1814-1815 doi:10.1073/pnas.1120528109
Erlebacher, A., Lukens, A. K., Glimcher, L. H. (2002) Intrinsic susceptibility of mouse trophoblasts to natural killer cell-mediated attack in vivo. Proceedings of the National Academy of Sciences, 99 (26) 16940-16945 doi:10.1073/pnas.222652199
Albright, J. W., Albright, J. F. (1983) Age-associated impairment of murine natural killer activity. Proceedings of the National Academy of Sciences, 80 (20) 6371-6375 doi:10.1073/pnas.80.20.6371
Vanherberghen, B., Andersson, K., Carlin, L. M., Nolte-`t Hoen, E. N. M., Williams, G. S., Hoglund, P., Davis, D. M. (2004) Human and murine inhibitory natural killer cell receptors transfer from natural killer cells to target cells. Proceedings of the National Academy of Sciences, 101 (48) 16873-16878 doi:10.1073/pnas.0406240101
 
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