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McDaniel, L. D., Schultz, R. A. (1992) Elevated sister chromatid exchange phenotype of Bloom syndrome cells is complemented by human chromosome 15. Proceedings of the National Academy of Sciences, 89 (17) 7968-7972 doi:10.1073/pnas.89.17.7968

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Reference TypeJournal (article/letter/editorial)
TitleElevated sister chromatid exchange phenotype of Bloom syndrome cells is complemented by human chromosome 15.
JournalProceedings of the National Academy of Sciences
AuthorsMcDaniel, L. D.Author
Schultz, R. A.Author
Year1992 (September 1)Volume89
Issue17
PublisherProceedings of the National Academy of Sciences
DOIdoi:10.1073/pnas.89.17.7968Search in ResearchGate
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Mindat Ref. ID1512773Long-form Identifiermindat:1:5:1512773:0
GUID0
Full ReferenceMcDaniel, L. D., Schultz, R. A. (1992) Elevated sister chromatid exchange phenotype of Bloom syndrome cells is complemented by human chromosome 15. Proceedings of the National Academy of Sciences, 89 (17) 7968-7972 doi:10.1073/pnas.89.17.7968
Plain TextMcDaniel, L. D., Schultz, R. A. (1992) Elevated sister chromatid exchange phenotype of Bloom syndrome cells is complemented by human chromosome 15. Proceedings of the National Academy of Sciences, 89 (17) 7968-7972 doi:10.1073/pnas.89.17.7968
In(1992, September) Proceedings of the National Academy of Sciences Vol. 89 (17) Proceedings of the National Academy of Sciences

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Flejter, W. L., McDaniel, L. D., Johns, D., Friedberg, E. C., Schultz, R. A. (1992) Correction of xeroderma pigmentosum complementation group D mutant cell phenotypes by chromosome and gene transfer: involvement of the human ERCC2 DNA repair gene. Proceedings of the National Academy of Sciences, 89 (1) 261-265 doi:10.1073/pnas.89.1.261
Ray, J. H., Louie, E., German, J. (1987) Different mutations are responsible for the elevated sister-chromatid exchange frequencies characteristic of Bloom's syndrome and hamster EM9 cells. Proceedings of the National Academy of Sciences, 84 (8) 2368-2371 doi:10.1073/pnas.84.8.2368
Latt, S. A. (1974) Sister Chromatid Exchanges, Indices of Human Chromosome Damage and Repair: Detection by Fluorescence and Induction by Mitomycin C. Proceedings of the National Academy of Sciences, 71 (8) 3162-3166 doi:10.1073/pnas.71.8.3162
Popescu, N. C., Amsbaugh, S. C., DiPaolo, J. A. (1980) Enhancement of N-methyl-N'-nitro-N-nitrosoguanidine transformation of Syrian hamster cells by a phorbol diester is independent of sister chromatid exchanges and chromosome aberrations. Proceedings of the National Academy of Sciences, 77 (12) 7282-7286 doi:10.1073/pnas.77.12.7282
Weinreb, A., Katzenberg, D. R., Gilmore, G. L., Birshtein, B. K. (1988) Site of unequal sister chromatid exchange contains a potential Z-DNA-forming tract. Proceedings of the National Academy of Sciences, 85 (2) 529-533 doi:10.1073/pnas.85.2.529
Fukuchi, K., Martin, G. M., Monnat, R. J. (1989) Mutator phenotype of Werner syndrome is characterized by extensive deletions. Proceedings of the National Academy of Sciences, 86 (15) 5893-5897 doi:10.1073/pnas.86.15.5893
Chaganti, R. S. K., Schonberg, S., German, J. (1974) A Manyfold Increase in Sister Chromatid Exchanges in Bloom's Syndrome Lymphocytes. Proceedings of the National Academy of Sciences, 71 (11) 4508-4512 doi:10.1073/pnas.71.11.4508
Warren, S. T., Schultz, R. A., Chang, C. C., Wade, M. H., Trosko, J. E. (1981) Elevated spontaneous mutation rate in Bloom syndrome fibroblasts. Proceedings of the National Academy of Sciences, 78 (5) 3133-3137 doi:10.1073/pnas.78.5.3133
Shiraishi, Y., Yosida, T. H., Sandberg, A. A. (1983) Analyses of bromodeoxyuridine-associated sister chromatid exchanges (SCEs) in Bloom syndrome based on cell fusion: single and twin SCEs in endoreduplication. Proceedings of the National Academy of Sciences, 80 (14) 4369-4373 doi:10.1073/pnas.80.14.4369
Tartof, K. D. (1974) Unequal Mitotic Sister Chromatid Exchange as the Mechanism of Ribosomal RNA Gene Magnification. Proceedings of the National Academy of Sciences, 71 (4) 1272-1276 doi:10.1073/pnas.71.4.1272
(1989) Correction: Mutator Phenotype of Werner Syndrome is Characterized by Extensive Deletions. Proceedings of the National Academy of Sciences, 86 (20) 7994 doi:10.1073/pnas.86.20.7994
 
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