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New, M. I., Wilson, R. C. (1999) Steroid disorders in children: Congenital adrenal hyperplasia and apparent mineralocorticoid excess. Proceedings of the National Academy of Sciences, 96 (22) 12790-12797 doi:10.1073/pnas.96.22.12790

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Reference TypeJournal (article/letter/editorial)
TitleSteroid disorders in children: Congenital adrenal hyperplasia and apparent mineralocorticoid excess
JournalProceedings of the National Academy of Sciences
AuthorsNew, M. I.Author
Wilson, R. C.Author
Year1999 (October 26)Volume96
Issue22
PublisherProceedings of the National Academy of Sciences
DOIdoi:10.1073/pnas.96.22.12790Search in ResearchGate
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Mindat Ref. ID1631016Long-form Identifiermindat:1:5:1631016:0
GUID0
Full ReferenceNew, M. I., Wilson, R. C. (1999) Steroid disorders in children: Congenital adrenal hyperplasia and apparent mineralocorticoid excess. Proceedings of the National Academy of Sciences, 96 (22) 12790-12797 doi:10.1073/pnas.96.22.12790
Plain TextNew, M. I., Wilson, R. C. (1999) Steroid disorders in children: Congenital adrenal hyperplasia and apparent mineralocorticoid excess. Proceedings of the National Academy of Sciences, 96 (22) 12790-12797 doi:10.1073/pnas.96.22.12790
In(1999, October) Proceedings of the National Academy of Sciences Vol. 96 (22) Proceedings of the National Academy of Sciences

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NEW, MARIA I. (2002) Hypertension in Congenital Adrenal Hyperplasia and Apparent Mineralocorticoid Excess. Annals of the New York Academy of Sciences, 970 (1) 145-154 doi:10.1111/j.1749-6632.2002.tb04420.x
(1987) Correction: P450XXI (steroid 21-hydroxylase) Gene Deletions are not Found in Family Studies of Congenital Adrenal Hyperplasia. Proceedings of the National Academy of Sciences, 84 (22) 8054 doi:10.1073/pnas.84.22.8054
White, P. C., New, M. I., Dupont, B. (1984) HLA-linked congenital adrenal hyperplasia results from a defective gene encoding a cytochrome P-450 specific for steroid 21-hydroxylation. Proceedings of the National Academy of Sciences, 81 (23) 7505-7509 doi:10.1073/pnas.81.23.7505
Harada, F., Kimura, A., Iwanaga, T., Shimozawa, K., Yata, J., Sasazuki, T. (1987) Gene conversion-like events cause steroid 21-hydroxylase deficiency in congenital adrenal hyperplasia. Proceedings of the National Academy of Sciences, 84 (22) 8091-8094 doi:10.1073/pnas.84.22.8091
New, M. I., Abraham, M., Gonzalez, B., Dumic, M., Razzaghy-Azar, M., Chitayat, D., Sun, L., Zaidi, M., Wilson, R. C., Yuen, T. (2013) Genotype-phenotype correlation in 1,507 families with congenital adrenal hyperplasia owing to 21-hydroxylase deficiency. Proceedings of the National Academy of Sciences, 110 (7) 2611-2616 doi:10.1073/pnas.1300057110
Haider, S., Islam, B., D'Atri, V., Sgobba, M., Poojari, C., Sun, L., Yuen, T., Zaidi, M., New, M. I. (2013) Structure-phenotype correlations of human CYP21A2 mutations in congenital adrenal hyperplasia. Proceedings of the National Academy of Sciences, 110 (7) 2605-2610 doi:10.1073/pnas.1221133110
Matteson, K. J., Phillips, J. A., Miller, W. L., Chung, B. C., Orlando, P. J., Frisch, H., Ferrandez, A., Burr, I. M. (1987) P450XXI (steroid 21-hydroxylase) gene deletions are not found in family studies of congenital adrenal hyperplasia. Proceedings of the National Academy of Sciences, 84 (16) 5858-5862 doi:10.1073/pnas.84.16.5858
LIN-SU, K., NEW, M. I. (2007) Effects of Adrenal Steroids on the Bone Metabolism of Children with Congenital Adrenal Hyperplasia. Annals of the New York Academy of Sciences, 1117 (1) 345-351 doi:10.1196/annals.1402.040
Curnow, K. M., Slutsker, L., Vitek, J., Cole, T., Speiser, P. W., New, M. I., White, P. C., Pascoe, L. (1993) Mutations in the CYP11B1 gene causing congenital adrenal hyperplasia and hypertension cluster in exons 6, 7, and 8. Proceedings of the National Academy of Sciences, 90 (10) 4552-4556 doi:10.1073/pnas.90.10.4552
(1999) Proceedings of the National Academy of Sciences, 96 (22) 12970 doi:10.1073/pnas.96.22.12970
White, P. C., New, M. I., Dupont, B. (1984) Cloning and expression of cDNA encoding a bovine adrenal cytochrome P-450 specific for steroid 21-hydroxylation. Proceedings of the National Academy of Sciences, 81 (7) 1986-1990 doi:10.1073/pnas.81.7.1986
 
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